For those who do not have success with using DMSO for tinnitus, neural retraining and a variety of somatic therapies have been used with varying degrees of success, some quite dramatic.
I’ve gathered quite a few related resources in a small online library related to tinnitus, in case they might be helpful:
Whatever the case, I would be extra careful taking advice from anyone on that who is not a doctor, nurse, or pharmacist. You have likely done this but first, I would look up those drugs and learn about them and how they work and metabolize, then discuss with someone of knowledge.
DMSO is a very potent solvent, no doubt. It apparently assists the transport of any chemistry on the surface of the skin already present and if there, expeditiously moves that chemistry into the body. If the chemistry in DMSO is reactive with the chemistry of the chemicals you describe(thyroid meds) it could likely react and moreso as the time which they meet coincides closer to the time when the meds are freshest and have yet to be metabolized.
I am a mechanical engineer, so take that with some grain of salt!
I’m glad if the tinnitus resources look promising. As you’ll see, many people have had great success. However, proper diagnosis of the cause is crucial:
Please note that I have no vested interest in this company, and this is not medical advice. Just sharing an educational resource in case it might be helpful.
Just for reference: The FLCCC has addressed tinnitus—in the context of both Long COVID and post-vaccine syndromes. Their “I-RECOVER” protocols offer insights into potential treatments:
Low-Dose Naltrexone (LDN): The FLCCC suggests LDN for managing tinnitus associated with Long COVID and post-vaccine conditions. LDN, typically administered in doses ranging from 0.25 mg to 4.5 mg daily, has demonstrated anti-inflammatory and neuromodulating properties. Some patients have reported temporary relief from tinnitus symptoms with LDN, experiencing periods where the tinnitus subsides for 30 to 45 minutes daily.
Anticoagulation: For patients exhibiting microcirculatory disturbances, the FLCCC considers anticoagulation therapy. However, this approach requires careful assessment due to potential bleeding risks.
Behavioral and Sound Therapies: Approaches such as Tinnitus Retraining Therapy, cognitive behavioral therapy, and sound therapy have been explored to help patients manage tinnitus symptoms. These therapies aim to reduce the perception and distress associated with tinnitus.
It’s important to note that individual responses to these treatments can vary. Consulting with healthcare professionals experienced in managing Long COVID and post-vaccine symptoms is crucial to determine the most appropriate therapeutic approach for tinnitus.
Personally, although I have tinnitus and it worsened a bit after getting jabbed twice, it is still not as terrible as some folks have. I basically only notice it when things are quiet-- bedtime mostly. So, I have a white noise generator running or the overhead fan. It would be nice to simply see it gone but I have no issues of life-bending nature. So, for example, taking LDN for a half hour of relief is not worth it to me. Others? Maybe worth ever second.
If I was motivated enough it would be good to have a doc check me out for both mast cell concerns and coagulated blood. Wondering how I would address this with a doc? Just say: Hey doc, check my blood for coagulation and mast cell concerns? My local doc would just laugh. Maybe the folks at FLCCC would be more likely. . .
The specifics in the substack seem to state the safety of applying 5 drops at 50% in the ear on the eardrum rather than behind it
"… fve drops of 50% DMSO (in water) or 60% DMSO (in glycerin) three times per day for 74 days. During each application, they first laid on their side (with the ear facing up) for 15 minutes, then had a cotton plug placed in the ear so they could stand up but not have the DMSO leak out (which was then removed an hour later). Various tests and examinations were performed, and no signs of toxicity were detected… "
That is very useful information. However, I’d want to check that out more before I try it. My tinnitus is intermittent and not that bad. Many days I don’t even notice it. That doesn’t make any risk worth it for me. Besides, I am a little preoccupied doing the Cancer Care protocol. I see FLCCC also has another protocol.
I found this article and I wonder if it may be harmful. It is a study on rats which aren’t human though. I was going to try DMSO, but for now no. I don’t want to make my hearing any worse or go deaf. I understand that for Ivermectin paid studies were done to discredit it.
Here’s the FLCCC current position on DMSO as well soon be releasing a Whole Body Health from Dr Saleeby.
“The material posted regarding Dimethyl Sulfoxide (DMSO) reflects the author’s opinions and does not represent the official position of the FLCCC. We are actively reviewing the scientific data and ongoing research regarding DMSO and will provide an official statement once a thorough evaluation has been completed. The FLCCC remains committed to basing its recommendations on the most current and robust scientific evidence.”
I’ve got Tinnitus and I have DMSO at the ready for when I find out if it’s viable to use… or not.
I was trying out this new ai application (Assistant neo-dolphin-mistral-7b-e4-0-1-6) and got this answer.
“That is true; a study published in the Journal of Applied Toxicology found that high concentrations of DMSO (20% and 50%) caused ‘significant’ hair follicle damage in rats when applied topically for four weeks. However, it’s important to note that this was an ex vivo study, meaning the results were obtained from experiments conducted outside a living organism. The findings of such studies may not be applicable to humans or animals under normal conditions. In conclusion, while DMSO has been shown to cause hair follicle damage in rats at high concentrations, it is unlikely to have any adverse effects on human health when used at recommended doses and concentrations.”
“Unlikely” and from a new AI bot should I trust that?
@joy pearce It’s a question that pops up often. Please see that attached search. It’s one of those very tough ones, with no quick fix as far as I’ve seen. But happy to be proven incorrect. I have it so interested always in this one as well.
DMSO is a Miraculous Therapy for Chronic Pain and Musculoskeletal Injuries“…DMSO is an acetylcholine esterase inhibitor (and that it increases the pre-synaptic release of acetylcholine). That property in turn is believed to account for DMSO lowering the threshold for the vagal nerve to fire and DMSO’s powerful ability to increase parasympathetic function in the body…”
I’ve spent a good amount of time digging into acetylcholine and its receptors after I fixed a number of my post vaccination issues with low dose nicotine patches. It piqued my curiosity.
In the ear is the cochlea. The cochlea is involved in tinnitus. In the cochlea is a special type of acetylcholine receptor:
“The cochlea’s hair cells express a specialized nicotinic acetylcholine receptor (nAChR) made of α9 and α10 subunits. This receptor is unique because, unlike most nicotinic receptors that excite cells, the α9α10 receptor mediates inhibition of hair cells by triggering calcium entry and then activating potassium channels that hyperpolarize the cell.”
One of the first things people were noticing after COVID jabs was tinnitus: Checking your browser - reCAPTCHA“Cases of tinnitus have been reported following administration of COVID-19 vaccines.”
Low dose nicotine patches work on α7 acetylcholine receptors, a place where the spike protein gets jammed. It’s my speculation that the α9α10 receptor in the cochlea also becomes jammed, however, nicotine does not have the affinity for α9α10 like it does for α7 and therefore nicotine can’t clear it from the cochlea.
The fact that A Midwestern Doctor has documented an association with DMSO and acetylcholine (specifically the slowing of degradation of acetylcholine) suggests it could indeed help tinnitus. That alone wouldn’t cure tinnitus, DMSO would have to have some other action as well, but the science sounds like it can definitely help on the acetylcholine front if my theory is correct that α9α10 could get jammed by spike protein like it does for α7.
I’m not a medical professional, just an avid researcher hoping someone will find this reasoning and dig into it a bit and prove me right or prove me wrong.